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Community Collective: Obesity
NMPD Series
Obstructive Sleep Apnea Series
A Quick Dose of CE
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Medical Learning Institute Inc
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About Us
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About MLI
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Community Collective: Obesity
NMPD Series
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A Quick Dose of CE
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IASP Post-Test Evaluation (P22081-01)
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1. Regarding the current theories about the pathophysiology of migraine:
It is primarily an intracranial vascular disorder with neurological manifestations due to dopamine excess.
It is primarily a neurological disorder with vascular inflammation due to serotonin depletion.
It is primarily a vascular disorder with neurogenic inflammation.
It is an immune-mediated neurological disorder with involvement of the intracranial vessels.
2. The most common presentation of acute back pain is related to:
Herniated disc
Osteoarthritis
Muscle/myofascial problem
Trauma
3. What is a common denominator for OA, CLBP, and migraine?
Age and gender-specific disorders
All may develop pain sensitization
Closely related to size of structures involved
4. Pain sensitization in OA can best be evaluated by:
Visual analogue scales
Quantitative sensory testing
Serological markers
5. All of the following are true regarding acute back pain except:
25% of patients with low back pain miss 10 days or work or greater annually
Facetogenic pain is often aggravated by flexion
No imaging/work up is needed for the first 6 weeks unless clear red flags are present
It is important to understand the psychological complexities patients face while attempting to diagnose the cause of their pain
6. A 26-year-old primiparous woman is referred to you by her obstetrician for unilateral headaches in the first trimester that are disabling her. Secondary headache disorders have been ruled out. The woman is concerned that she cannot enjoy the rest of her pregnancy and asks for prophylactic options. What would you suggest?
NSAIDs, since these help with acute headaches as well as to prevent them
Ditans, since these have been shown to be safe
Beta blockers, metoprolol, propranolol as they are relatively safe in migraine in pregnancy
Ergot alkaloids for prophylaxis and IV valproate for the acute attacks
7. Where do NSAIDs interact with the pain system?
Exclusively at the peripheral level
Exclusively at the central level
May interact with mechanisms at both levels
8. Managing OA pain flare ups is best treated by:
Physiotherapy
Exercise
Combination of pain education, physiotherapy and exercise
Short term use of NSAIDs +/- acetaminophen
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